Study · Nature communications · 2025Conflicts declared

Chili pepper compound, gut inflammation and short stature in children

What it found

Children with idiopathic short stature had higher levels of a tiny RNA piece called miR-17-3p in their blood exosomes and stool, and a capsaicin-rich diet caused mild gut inflammation and raised the same marker in rats, which grew shorter.

The work links a spicy diet to a possible pathway affecting growth, though the human data are observational.

What they found

Studies in people

Higher miR-17-3p in children

Blood exosomes from children with idiopathic short stature had higher levels of miR-17-3p than those from healthy children. The authors report this marker distinguished the two groups with high sensitivity and specificity.

Stool inflammation markers

Stool samples from children with idiopathic short stature had more miR-17-3p and more of the inflammatory marker TNF-alpha than samples from healthy children.

Animal and lab studies

Capsaicin diet in rats

Rats fed a capsaicin-rich diet for 28 days grew shorter and lighter than rats on a capsaicin-free diet, even though they ate similar amounts after the first few days. Their growth plates showed less cartilage cell growth and bone formation.

Gut inflammation in rats

The capsaicin-fed rats had mild inflammation in their small intestine and colon, with damage to the villi and higher levels of inflammatory cytokines. The authors call this microinflammation.

miR-17-3p pathway

In lab tests, capsaicin activated the NF-kB signaling pathway in intestinal cells, which raised miR-17-3p. This miR-17-3p then suppressed the ZNF148/SOS1 axis, reducing RAS/ERK and PI3K/AKT signaling needed for cartilage cell growth.

Engineered exosome treatment

In rats, exosomes engineered to target growth plate cartilage and carry a miR-17-3p blocker plus growth hormone restored growth plate function and improved height. The authors say this approach needs more safety testing.

What the authors conclude

“These findings uncover a diet-driven exosome axis underlying ISS and suggest new therapeutic strategies for children in high-capsaicin regions.”
Wang Y, Wu Z, Yuan J, et al, 2025

Also in their conclusions

  • They say a capsaicin-rich diet can cause mild gut inflammation that activates NF-kB and raises plasma exosomal miR-17-3p, which inhibits bone growth and contributes to growth hormone insensitivity in children with idiopathic short stature.
  • They call for more attention to the impact of capsaicin-rich diets on children's growth, especially in high-risk groups and those receiving growth hormone therapy.
  • They suggest that engineered exosomes delivering miR-17-3p siRNA and growth hormone to the growth plate could be a promising treatment strategy.

How it was done

The authors wanted to understand what causes idiopathic short stature, since most cases have no known reason and many children respond poorly to growth hormone treatment. They noticed that regions of China with high chili pepper consumption also report more cases of idiopathic short stature.

They compared blood exosomes from 106 children with idiopathic short stature and 106 matched healthy children, and looked at stool samples from both groups. They also fed immature rats a diet with 0.01% capsaicin for 28 days and studied their growth plates, intestines and blood exosomes.

What it can’t tell you

  • The human part of this study compared children at one point in time, so it can show a link between diet, gut inflammation and short stature but cannot prove that chili pepper causes it.
  • The rat experiments used a specific capsaicin dose and cannot show what happens in children eating spicy food over many years.
  • The authors note that the engineered exosome treatment needs more study before its full safety profile is known.

The paper

Title
Capsaicin diet drives gut inflammation and exosomal miR-17-3p elevation in idiopathic short stature
Type
Study
Evidence
Studies in people · animals · lab
Summarised from
Full text
Cite
Wang Y, Wu Z, Yuan J, et al (2025). Capsaicin diet drives gut inflammation and exosomal miR-17-3p elevation in idiopathic short stature. Nature communications. doi:10.1038/s41467-025-67883-2Free full textPubMed 41469526DOI

Summary written 26 Sep 2026. Check it against the paper before it changes what you eat. How we summarise papers · Report an error